High hepatitis B virus (HBV) DNA viral load as the most important risk factor for HBV reactivation in patients positive for HBV surface antigen undergoing autologous hematopoietic cell transplantation Clinical Trials & Observations George K. In the process of HBV clearance, virus may infect lymphocytes by way of infecting stem cells, phagocytic function of lymphocytes, or the cytotoxic effect mediated by hepatic surface-re ceptors.
Occult HBV, with loss of HBsAg Chronic Inactive HBV Chronic HBV, e-Ag positive Chronic HBV, e-Ag negative Reactivation of Chronic HBV Test is only needed if patient is HBsAg positive or there is suspicion of occult HBV. To compare serum HBV DNA levels in different states of hepatitis B, 47 sera of patients with HBeAg-positive chronic hepatitis B, 4 sera of patients with HBeAg-negative chronic hepatitis B, 40 samples of patients after HBeAg seroconversion during alpha interferon treatment, 57 sera of inactive HBsAg. When daily antiviral pills (either tenofovir or entecavir) are prescribed, doctors measure your HBV DNA to see if the drug is working to reduce your viral load. HBsAg: Neg: Pos: Pos: Pos: Pos: HBV surface antibody (HBsAb) Neg: Neg: Neg: Neg: Neg: HBV core antibody IgG (HBcAb IgG) Pos.
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H e p a ti ti s B S u r fa c e A n ti b o d y (HbsAb or Anti-HBs) - The "surface antibody" is formed in response to the hepatitis B virus. Because anti-HCV testing prior to age 18 months may be falsely positive due to detection of passively acquired maternal antibody, testing prior to age 18 months should consist of HCV RNA testing 1. Includes full text Arabic, French, German, Japanese, Portuguese, Spanish, simplified-PRC Chinese, traditional Chinese, and Korean with summary translations.